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Download e-book for iPad: Cardiovascular Physiology 8/E by David Mohrman, Lois Heller

By David Mohrman, Lois Heller

ISBN-10: 0071793119

ISBN-13: 9780071793117

The most sensible cardiovascular body structure textual content for USMLE and examination review

Cardiovascular Physiology is a concise and relaxing method that you should achieve a basic wisdom of the fundamental working ideas of the intact cardiovascular procedure and the way these ideas follow to scientific medication. Succinct yet thorough, it makes a speciality of the evidence and ideas you need to recognize to get a fantastic "big photo" review of ways the cardiovascular procedure operates in common and irregular events. No different textual content will end up extra beneficial in bettering your skill to guage the myriad new info you may be uncovered to all through your profession, than Cardiovascular Physiology.


  • NEW incorporates a "Perspectives" part in each one bankruptcy that identifies vital unresolved matters
  • Clarifies the main points of physiologic mechanisms and their function in pathologic states
  • Links cardiovascular body structure to analysis and remedy
  • Summarizes key ideas on the finish of every bankruptcy
  • Highlights must-know details with bankruptcy ambitions
  • Reinforces studying with research questions on the finish of every chapter

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Extra info for Cardiovascular Physiology 8/E

Example text

CHARACTERISTICS OF CARDIAC MUSCLE CELLS I 39 tension development11; {2) a complex internal compartmentation of the cytoplasm by an intracellular membrane system called the sarcoplasmic reticulum {SR), which actively sequesters calcium during the diastolic interval with the help of the sarco/ endoplasmic reticulum Ca2+-ATPase (SERCA) and calcium-binding storage pro­ teins, the most abundant of which is calsequestrin; (3) regularly spaced, extensive invaginations of the cell membrane (sarcolemma), called T tubules, which appear to be connected to parts of the SR {"junctional" SR) by dense strands {"feet") and which carry the action potential signal to the inner parts of the cell; and {4) a large number of mitochondria that provide the oxidative phosphorylation pathways needed to ensure a ready supply of adenosine triphosphate {ATP) to meet the high metabolic needs of the cardiac muscle.

Therefore, when K+ diffuses out of a cell, it creates an electrical potential across the membrane that tends to attract it back into the cell. There exists one membrane potential called the potassium equilibrium potential at which the electrical forces tending to pull K+ into the cell exactly balance the concentration forces tending to drive K+ out. When the membrane potential has this value, there is no net movement of K+ across the membrane. 5 mY = z Iog [ X' ] inside 10 [ X' ] outside 26 I CHAPTER TWO and rapidly (essentially instantaneously) develop the potassium equilibrium poten­ tial.

The force that a muscle produces during an isometric contraction indicates its maximum ability to develop tension. At the other extreme, activating an unrestrained muscle causes it to shorten without force development because it has nothing to develop force against. This type of contraction is called an isotonic ("fixed tension") contraction. Under such 131he action of these pumps is regulated by the protein phospholamban. When this protein is phosphory­ lated (eg, by the action of norepinephrine), the rate of Ca2+ resequestration by the sarcoplasmic reticulum is increased and the rate of relaxation is enhanced.

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Cardiovascular Physiology 8/E by David Mohrman, Lois Heller

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