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Cardiovascular Hemodynamics for the Clinician by George A. Stouffer PDF

By George A. Stouffer

ISBN-10: 1405169176

ISBN-13: 9781405169172

Now you've a accountable consultant to the sensible software of hemodynamics. This concise instruction manual can help either practising and potential clinicians larger comprehend and interpret the hemodynamic facts used to make particular diagnoses and video display ongoing therapy.Written from the viewpoint of a clinician, this handy paperback opens with an outline of the fundamentals of hemodynamics, then devotes chapters to express illness states. issues include:• coronary artery disorder• cardiomyopathies• valve illness• arrhythmias• pericardial diseaseNumerous strain tracings in the course of the e-book make stronger the textual content via demonstrating what you can find in day-by-day perform. To extract as a lot worthwhile info as attainable from the hemodynamic facts received out of your sufferers, make sure to seek advice Cardiovascular Hemodynamics for the Clinician.

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Extra info for Cardiovascular Hemodynamics for the Clinician

Example text

Case study answers 1. a. SV = CO/HR = 3500 ml/min/120 bpm = 29 ml. The patient’s stroke volume is 29 mL. Chapter 2 PA catheter placement 35 b. 5 = 1051 dynes/ sec/cm5 . c. Preload is reflected by CVP =10 mm Hg and PCWP = 16 mm Hg with an afterload (SVR) of 1051 dynes/sec/cm5 . Cardiac output is low despite reasonable preload suggesting LV dysfunction (possibly due to a cardiac contusion suffered at the time of the accident). An echocardiogram would be useful. Giving IV fluids to increase PCWP might increase cardiac output (at the risk of worsening oxygenation) and/or an inotrope such as Dobutamine might be used to increase contractility.

13 Schematics of pressure volume loops in various disease states. In aortic stenosis ((a); dotted line is normal ventricle), note the marked increase in afterload, minimal increase in preload and increased contractility. Chronic aortic insufficiency ((b); dotted line, normal ventricle; dashed line, acute AI; full line, chronic AI) is characterized by marked increase in preload with a smaller increase in afterload and decreased contractility. Acute aortic insufficiency manifests as increased preload with minimal change in afterload or contractility.

Despite an increase in CVP to 10 mm Hg after fluid resuscitation and the addition of two vasopressors, her blood pressure does not improve. Her chest x-ray shows diffuse pulmonary edema. 5 L/min a. Based on the information available, is the primary etiology of the pulmonary edema cardiogenic or noncardiogenic? 3. You are working in the CCU when a 62 year old female is admitted with the diagnosis of cardiogenic shock. The patient became extremely fatigued 48 hours ago but only recently came to the ER when she became very short of breath.

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Cardiovascular Hemodynamics for the Clinician by George A. Stouffer


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